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Differential therapeutic effects of non-carrier substance P antagonists on heart failure in rats.
We examined the therapeutic effects of non-carrier substance P antagonists on heart failure (HF) in rats. We administered a non-carrier substance P antagonist (D-Arg1, D-Pro2, D-Trp7,9, Leu11-substance P [S-95715]) to rats in the presence or absence of HF induced by an endotoxin. S-95715 (0.5 mg/kg twice a day, for 7 days) also was administered to rats without HF. Cardiac function in rats that received S-95715 alone was similar to that of rats without HF. Cardiac performance in rats without HF that received S-95715 (0.5 mg/kg twice a day for 7 days) was improved and the elevated plasma epinephrine levels were reduced. Plasma norepinephrine levels were suppressed by S-95715 (0.5 mg/kg twice a day for 7 days) in a dose-dependent manner. S-95715 (0.5 mg/kg twice a day for 7 days) did not improve the impaired cardiac function in rats with HF. Plasma norepinephrine and epinephrine levels were elevated to a lesser degree in rats with HF than in rats without HF. These results suggest that non-carrier substance P antagonists may be more useful than the carrier molecule in suppressing sympathetic overactivity

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